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1. CTCF shapes chromatin by multiple mechanisms: the impact of 20 years of CTCF research on understanding the workings of chromatin


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2. Acetylation of histone tails reduces the association with both p66-proteins


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3. Expression of p66α or p66β increases MBD2-mediated repression


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4. P66α- or p66β-mediated repression acts downstream of MBD2


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5. P66α or p66β requires the paralogous p66 partner for repression


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6. Both p66α and p66β interact with histone tails


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7. Knock-down of endogenous p66α or p66β decreases MBD2-mediated repression


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8. K149 of p66α is required for the MBD2 interaction as well as for the MBD2-mediated repression


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9. Nuclear distribution of p66α and p66β depends on CR2 and K149 for p66α


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10. Nuclear distribution of p66α and p66β depends on the presence of MBD2


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