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1. Chronic delayed-type hypersensitivity reaction as a means to treat alopecia areata

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http://www.ncbi.nlm.nih.gov/pmc/articles/PMC1808967
http://www.ncbi.nlm.nih.gov/pubmed/15008971  
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2. The pathogenesis of alopecia areata in rodent models.


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3. The functional relevance of the type 1 cytokines IFN-gamma and IL-2 in alopecia areata of C3H/HeJ mice.


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4. Interferon-gamma-deficient mice are resistant to the development of alopecia areata.


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5. Treatment with an anti-CD44v10-specific antibody inhibits the onset of alopecia areata in C3H/HeJ mice [In Process Citation]


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6. Resistance to alopecia areata in C3H/HeJ mice is associated with increased expression of regulatory cytokines and a failure to recruit CD4+ and CD8+ cells.


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7. Successful treatment of alopecia areata-like hair loss with the contact sensitizer squaric acid dibutylester (SADBE) in C3H/HeJ mice.


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8. Interleukin-10-deficient mice are less susceptible to the induction of alopecia areata.


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9. Treatment of alopecia areata in C3H/HeJ mice with the topical immunosuppressant FK506 (Tacrolimus).


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10. Dietary soy oil content and soy-derived phytoestrogen genistein increase resistance to alopecia areata onset in C3H/HeJ mice.


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