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1. Advances in Brief The Expression of the Antiproliferative Gene ZAC Is Lost or Highly Reduced in

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2. Title not available

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3. Transcriptome analysis of MENX-associated rat pituitary adenomas identifies novel molecular mechanisms involved in the pathogenesis of human pituitary gonadotroph adenomas

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4. Endocrine-Related Cancer (2008) 15 583–596 Efficacy of a dopamine–somatostatin

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5. Study on transcription factors involved in the pathogenesis of pituitary adenomas


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6. FKBPs and the Akt/mTOR pathway

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http://www.ncbi.nlm.nih.gov/pmc/articles/PMC
http://www.ncbi.nlm.nih.gov/pubmed/23839048  
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7. Loss of function of the tumor suppressor DKC1 perturbs p27 translation control and contributes to pituitary tumorigenesis

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http://www.ncbi.nlm.nih.gov/pmc/articles/PMC2913864
http://www.ncbi.nlm.nih.gov/pubmed/20587522  
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8. c-Src Regulates Akt Signaling in Response to Ghrelin via β-Arrestin Signaling-Independent and -Dependent Mechanisms

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http://www.ncbi.nlm.nih.gov/pmc/articles/PMC2650409
http://www.ncbi.nlm.nih.gov/pubmed/19262695  
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9. Retinoic acid prevents experimental Cushing syndrome

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10. The SHP-1 protein tyrosine phosphatase negatively modulates Akt signaling in the ghrelin/GHSR1a system

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http://www.ncbi.nlm.nih.gov/pmc/articles/PMC3204078
http://www.ncbi.nlm.nih.gov/pubmed/21900501  
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