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1. Klinische, immunologische und molekulargenetische Differenzierung von Patienten mit autosomal-dominantem Hyper-IgE-Syndrom (HIES) und Patienten mit Verdacht auf HIES


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2. Abnormalities in Interferon-G and Toll-Like Receptor Signaling Suggest Different Monogenetic Defects Underlying Paracoccidioidomycosis Disease


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