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1. Hyaluronan Is Not a Ligand but a Regulator of Toll-Like Receptor Signaling in Mesangial Cells: Role of Extracellular Matrix in Innate Immunity

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2. Anti-GBM Glomerulonephritis Involves IL-1 but Is Independent of NLRP3/ASC Inflammasome-Mediated Activation of Caspase-1

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3. Funktion der Pathogen-Erkennungsrezeptoren des angeborenen Immunsystems auf Mesangialzellen bei der experimentellen Immunkomplex-Glomerulonephritis


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4. Hyaluronan Is Not a Ligand but a Regulator of Toll-Like Receptor Signaling in Mesangial Cells: Role of Extracellular Matrix in Innate Immunity

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http://www.ncbi.nlm.nih.gov/pmc/articles/PMC
http://www.ncbi.nlm.nih.gov/pubmed/24967246  
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5. Changes in macrophage phenotype as the immune response evolves

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http://www.ncbi.nlm.nih.gov/pmc/articles/PMC
http://www.ncbi.nlm.nih.gov/pubmed/23747023  
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6. Renal Failure Increases Cardiac Histone H3 Acetylation, Dimethylation, and Phosphorylation and the Induction of Cardiomyopathy-Related Genes in Type 2 Diabetes

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http://www.ncbi.nlm.nih.gov/pmc/articles/PMC2832129
http://www.ncbi.nlm.nih.gov/pubmed/20075197  
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7. Mdm2 Promotes Systemic Lupus Erythematosus and Lupus Nephritis

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http://www.ncbi.nlm.nih.gov/pmc/articles/PMC3279995
http://www.ncbi.nlm.nih.gov/pubmed/21949095  
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8. Viral RNA and DNA Trigger Common Antiviral Responses in Mesangial Cells

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http://www.ncbi.nlm.nih.gov/pmc/articles/PMC2736778
http://www.ncbi.nlm.nih.gov/pubmed/19713315  
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9. Progressive glomerulosclerosis in type 2 diabetes is associated with renal histone H3K9 and H3K23 acetylation, H3K4 dimethylation and phosphorylation at serine 10


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10. Progressive glomerulosclerosis in type 2 diabetes is associated with renal histone H3K9 and H3K23 acetylation, H3K4 dimethylation and phosphorylation at serine 10


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