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1. The two-pore channel TPCN2 mediates NAADP-dependent Ca2+-release from lysosomal stores


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2. Klonierung und funktionelle Charakterisierung neuer Vertreter der Superfamilie spannungsaktivierter Kationenkanäle


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3. Kv3.1 channels stimulate adult neural precursor cell proliferation and neuronal differentiation

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http://www.ncbi.nlm.nih.gov/pmc/articles/PMC
http://www.ncbi.nlm.nih.gov/pubmed/23478135  
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4. Kv3.1 channels stimulate adult neural precursor cell proliferation and neuronal differentiation


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5. Switching off calcium-dependent inactivation in l-type calcium channels by an autoinhibitory domain

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http://www.ncbi.nlm.nih.gov/pmc/articles/PMC1622877
http://www.ncbi.nlm.nih.gov/pubmed/17028172  
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6. Differential Cav2.1 and Cav2.3 channel inhibition by baclofen and α-conotoxin Vc1.1 via GABAB receptor activation

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http://www.ncbi.nlm.nih.gov/pmc/articles/PMC
http://www.ncbi.nlm.nih.gov/pubmed/24688019  
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7. Calmodulin Is a Functional Regulator of Cav1.4 L-type Ca2+ Channels*

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http://www.ncbi.nlm.nih.gov/pmc/articles/PMC2785611
http://www.ncbi.nlm.nih.gov/pubmed/19717559  
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8. γ-Aminobutyric Acid Type B (GABAB) Receptor Expression Is Needed for Inhibition of N-type (Cav2.2) Calcium Channels by Analgesic α-Conotoxins*

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http://www.ncbi.nlm.nih.gov/pmc/articles/PMC3390670
http://www.ncbi.nlm.nih.gov/pubmed/22613715  
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9. The two-pore channel TPCN2 mediates NAADP-dependent Ca2+-release from lysosomal stores

URL:
http://www.ncbi.nlm.nih.gov/pmc/articles/PMC2719734
http://www.ncbi.nlm.nih.gov/pubmed/19557428  
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10. Identifying Key Amino Acid Residues That Affect α-Conotoxin AuIB Inhibition of α3β4 Nicotinic Acetylcholine Receptors*

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http://www.ncbi.nlm.nih.gov/pmc/articles/PMC
http://www.ncbi.nlm.nih.gov/pubmed/24100032  
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