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1. A role for elevated H-2 antigen expression in resistance to neoplasia caused by radiation-induced leukemia virus. Enhancement of effective tumor surveillance by killer lymphocytes

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2. IDENTIFICATION OF A 36,000-MOLECULAR WEIGHT, GAG-RELATED PHOSPHOPROTEIN IN LYMPHOMA CELLS TRANSFORMED BY RADIATION LEUKEMIA VIRUS

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3. IDENTIFICATION OF A 36,000-MOLECULAR WEIGHT, GAG-RELATED PHOSPHOPROTEIN IN LYMPHOMA CELLS TRANSFORMED BY RADIATION LEUKEMIA VIRUS

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4. Open Access

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5. Brief Definitive Report EXPRESSION OF A SINGLE MAJOR HISTOCOMPATIBILITY COMPLEX LOCUS CONTROLS THE IMMUNE RESPONSE TO POLY-L-(TYROSINE

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6. Genetic control of radiation leukemia virus-induced tumorigenesis. I. Role of the major murine histocompatibility complex

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7. GENETIC CONTROL OF CELL-MEDIATED RESPONSIVENESS TO AN AKR TUMOR-ASSOCIATED ANTIGEN Mapping of the Locus Involved to the I Region of the H-2 Complex*

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8. Interactions Between Laminin Receptor and the Cytoskeleton During Translation and Cell Motility

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9. GENETICS OF SUSCEPTIBILITY TO RADIATION-INDUCED LEUKEMIA Mapping of Genes Involved to Chromosomes 1, 2, and 4, and Implications for a Viral Etiology in the Disease*

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10. A Novel cDNA Transcript Expressed in Fractionated X-lrradiation-induced Murine Thymomas’

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